Expert Views on HPV Infection

© 2019 by the authors; CC BY license

human papillomavirus; squamo-columnar junctions; (pre)neoplastic lesions; human papillomavirus; hypoxia; cervical cancer; head and neck cancer; senescence; metabolism; mTOR; therapy; human papillomavirus; papillomaviruses; oncogenic virus; cancer; HPV E6; HPV E7; hTERT; telomerase; betapapillomavirus, extracellular matrix; invasion; cancer initiating cells; wound healing; squamous cell carcinoma; head and neck cancer; human papillomavirus; human papillomavirus 16; HPV16; The Cancer Genome Atlas; oral keratinocytes; integration; episomal; mixed; HPV; somatic mutation; cervical cancer; APOBEC; significantly mutated gene; copy number variation; chromosomal instability; head and neck cancer; integration; exosomes; microvesicles; apoptotic bodies; papillomavirus; horizontal gene transfer; cell-to-cell communication; carcinogenesis; human papillomavirus; stroma; tumor microenvironment; cervical cancer; paracrine signaling; epithelial–stromal interactions; HPV; stem cells; cellular plasticity; HPV; persistent infection; cervical cancer; therapeutics; vaccines; episome maintenance; E2 protein; Keywords: papillomavirus; human papillomavirus (HPV); apolipoprotein B messenger RNA-editing, enzyme-catalytic, polypeptide-like 3 (APOBEC3); innate immunity; virus restriction; cancer mutagenesis; cancer progression; somatic mutation; virus evolution; papillomaviruses; protein domains; structural domains; origin; human papillomavirus; infection; epithelial differentiation; gene regulation; RNA processing; the mouse papillomavirus; tissue tropism; anogenital; oral infection; skin carcinoma; pathogenesis; innate immunity; adaptive immunity; RNA sequencing; host defense; Human papillomavirus; HPV; cervical cancer; epigenetics; histone; human papillomavirus; cervical cancer; skin cancer; epidermodysplasia verruciformis; immune evasion; chronic inflammation; IL-6; JAK-STAT3; immunotherapy; immunoscore; virus; HPV; cell cycle; differentiation; replication; DNA damage response; papillomavirus; latency; cervical cancer; clearance; persistence; latency; chronic; meta-population; fertility; virome; warts; cancer; evolution; human papillomavirus; HPV16; L2; subcellular trafficking; mitosis; transmembrane domain; translocation; membrane penetration; toxin; fusion peptide; gamma secretase; retromer; papillomaviruses; oncoproteins; structure; X-ray; NMR; virus-host interactomics; human papillomavirus; Fanconi anemia; orphan disease; squamous cell carcinoma; inherited cancer susceptibility; HPV carcinogenesis; HPV epidemiology; HPV genomics; viral–host interactions; HPV16; n/a