Dear Colleagues,

Autophagy is an evolutionary, highly-conserved, catabolic process that provides metabolic sufficiency by recycling bio-products, such as fatty acids, amino acids and nucleotides. After more than 50 years since the first report describing autophagic process, we now know that autophagy is more than a catabolic process, it is an essential cellular quality control mechanism that ensures cellular homeostasis during damaging insults, including those that damage organelles, accumulate protein aggregates, or lead to pathogen invasion. Deregulation of autophagy has been observed in many human diseases, and it is responsible for resistance to therapy in certain contexts. Further understanding of how autophagy is triggered or deregulated and how that contributes to disease pathogenesis is essential for developing novel treatment strategies for autophagy-related diseases.

Please join us in presenting this Special Issue that reveals novel evidence supporting the importance of autophagy in pathological contexts.

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• Autophagy
• Chaperon-mediated Autophagy
• Mitophagy
• Lipophagy
• Reticulophagy and Ribophagy
• Pexophagy
• Auto-inflammatory Diseases
• Immune Diseases
• Infection
• Cancer
• Neurodegenerative Diseases
• Aging