Reprint
Research of Pathogenesis and Novel Therapeutics in Arthritis
Edited by
June 2019
366 pages
- ISBN978-3-03897-065-1 (Paperback)
- ISBN978-3-03897-066-8 (PDF)
This is a Reprint of the Special Issue Research of Pathogenesis and Novel Therapeutics in Arthritis that was published in
Biology & Life Sciences
Chemistry & Materials Science
Medicine & Pharmacology
Summary
Arthritis has a high prevalence globally and includes over 100 different types, the most common of which are rheumatoid arthritis, osteoarthritis, psoriatic arthritis, and inflammatory arthritis. The exact etiology of arthritis remains unclear and no cure exists. Anti-inflammatory drugs are commonly used in the treatment of arthritis but are associated with significant side effects. Novel modes of therapy and additional prognostic biomarkers are urgently needed for arthritis patients. This book summarizes and discusses the global picture of the current understanding of arthritis.
Format
- Paperback
License and Copyright
© 2019 by the authors; CC BY-NC-ND license
Keywords
biosimilars; Th9 lymphocytes; rheumatoid arthritis; infliximab; rheumatoid arthritis; bone erosion; osteoblasts; next-generation sequencing; bioinformatics; microRNA; messenger RNA; osteoarthritis; cell signaling; IL1β; WNT; antagonists; computational modeling; nitric oxide; clodronate; gene expression; osteoarthritis; progenitor cells; SOX9; spondyloarthropathies; inflammation; mesenchymal stem cells; visfatin; IL-6; TNF-α; osteoarthritis; miR-199a-5p; Epstein-Barr virus; glycoprotein 42; rheumatoid arthritis; shared epitope; triptolide; rheumatoid arthritis; basic research; clinical translation; osteoarthritis (OA); articular cartilage; molecular pathology; therapeutics; rheumatoid arthritis; antibodies; collagen; glycosylation; disease pathways; therapy; experimental arthritis; TNFα; etanercept; infliximab; adalimumab; certolizumab pegol; golimumab; rheumatoid arthritis; therapeutic antibody; structure; fraxinellone; collagen-induced arthritis; rheumatoid arthritis; inflammatory arthritis; osteoclastogenesis; sclareol; rheumatoid arthritis; synovial cell; collagen; mice; cytokines; Th17; MAPK; arthritis; osteoarthritis; rheumatoid arthritis; small-molecule inhibitor; chondrocytes; tumor necrosis factor-alpha; inflammation; rheumatoid arthritis; osteoarthritis; angiogenesis; cytokines; chemokines; early osteoarthritis; articular cartilage; proliferation; fibroblast growth factor 2; mitogen activated protein kinase; transforming growth factor β; SMA- and MAD-related protein; interleukin; nuclear factor kappa B; miRNA; adjuvant arthritis; arthritis; biomarkers; celastrol; inflammation; microRNA; miRNA; rat; rheumatoid arthritis; Traditional Chinese medicine; tripterine; triterpenoid; spinal fusion; biological; osteoblast; osteoclast; bisphosphonate; parathyroid hormone; bone morphogenetic protein; receptor activator of nuclear factor κB; stem cell; drug delivery system; anticitrullinated peptide antibodies; antirheumatic drug; autoimmune; disease-modifying; immunology; pathology; rheumatoid factor; rheumatoid arthritis; osteoarthritis; adipokines; obesity; rheumatoid arthritis; osteoarthritis; anti-arthritis; biomarkers