**About the Special Issue Editors**

**Elisabeth Deindl** graduated from the ZMBH in Heidelberg, Germany, where she worked on hepatitis B viruses. Thereafter, she joined the lab of Wolfgang Schaper at the Max-Planck-Institute in Bad Nauheim, where she started to decipher the molecular mechanisms of arteriogenesis. After a short detour to study stem cells, she again focused on arteriogenesis, and became a leading expert in her field. By using a peripheral model of arteriogenesis, she demonstrated that collateral artery growth is a matter of innate immunity and presents a blueprint for sterile inflammation, which is locally triggered by extracellular RNA.

**Paul Quax** completed his PhD at the University of Leiden, the Netherlands, on the role of plasminogen activators in tissue remodeling. He continued to work on this topic in relation to vascular remodeling, first at the Gaubius Laboratory TNO, and later at the Leiden University Medical Center, as a professor in experimental vascular medicine. His interest in arteriogenesis has been driven by the lack of therapeutic options for patients with peripheral arterial disease. The topics of his research are therapeutic arteriogenesis and angiogenesis induced by gene therapy, growth factors, modulation of inflammatory and immune response, the modulation of microRNAs, and other noncoding RNAs in small animal models.
