**5. Conclusions**

αM's protective effects in CDDP-induced toxicity in LLC-PK1 are mostly attributable to mitochondrial mass and function preservation.

**Author Contributions:** Conceptualization, L.M.R.-F.; methodology, L.M.R.-F.; formal analysis, L.M.R.-F. and O.E.A.-T.; investigation, L.M.R.-F., O.E.A.-T. and S.H.A.-R.; resources, I.R. and J.P.-C.; writing—original draft preparation, L.M.R.-F.; writing—review and editing L.M.R.-F., O.E.A.-T., S.H.A.-R. and J.P.-C.; supervision, J.P.-C.; funding acquisition, E.T. and J.P.-C.

**Funding:** This work was supported by Consejo Nacional de Ciencia y Tecnología (CONACyT, México Grant No. AI-S-7495); Programa de Apoyo a Proyecto de Investigación e Innovación Tecnológica (PAPIIT, UNAM, Mexico, Grants IN201316 and IN202219); Programa de Apoyo a la Investigación y el Posgrado (PAIP, Mexico, Grant No. 5000-9105); and Fondos del Gasto Directo autorizado a la Subdirección de Investigación Básica, Instituto Nacional de Cardiología Ignacio Chávez. L.M.R.F is a doctoral student from Programa de Maestría y Doctorado en Ciencias Bioquímicas, Universidad Nacional Autónoma de México (UNAM) and received a fellowship from CONACyT.

**Acknowledgments:** We thank Elena Martínez-Klimova for her assistance on the review of the present text.

**Conflicts of Interest:** The authors declare no conflict of interest.
