Reprint
Recent Advances in Scar Biology
Edited by
November 2018
202 pages
- ISBN978-3-03897-398-0 (Paperback)
- ISBN978-3-03897-399-7 (PDF)
This is a Reprint of the Special Issue Recent Advances in Scar Biology that was published in
Biology & Life Sciences
Chemistry & Materials Science
Medicine & Pharmacology
Summary
Scars develop in the final stage of wound healing. Wound healing and scarring involve complex biological pathways, and the exact mechanisms by which they are initiated, evolved, and regulated remain to be fully elucidated. Scarless wound healing is a major goal of medical science. To achieve this goal, it is necessary to elucidate the relevant clinical, histopathological, and molecular manifestations of scars, and to understand how these manifestations relate to each other.
This Special Issue covers a selection of recent research topics and current review articles in the field of scar research for all kinds of tissues and organs.
Format
- Paperback
License and Copyright
© 2019 by the authors; CC BY license
Keywords
trabeculectomy; glaucoma; gelatin hydrogel; transforming growth factor-β; beagles; polydeoxyribonucleotide; cicatrix, inflammation; wounds and injuries; rats; second harmonic generation; nonlinear optical microscopy; scar tissue; fibrillar collagen; skin; lung; vessels; image analysis; burn; wound healing; wound extract; granulation tissue; endothelial cell; fibroblast; ASC; skin; focal adhesion kinase; keratinocyte; mechanotransduction; extracellular matrix; single-cell transcriptional analysis; skin fibrosis; hypertrophic scar; transcriptomics; estrogen; estrogen receptor; wound healing; protease-activated receptor 2; transient receptor potential vanniloid 3; thymic stromal lymphopoietin; post-burn pruritus; keloid; cryotherapy; fibrosis; matrix metallopeptidase 9; transforming growth factor β 1; split-thickness skin grafting; cultured epithelial autografts (CEA); assessment of scar quality; generalized estimating equation (GEE); generalized linear mixed model (GLMM); keloid; hypertrophic scar; fibroblast; HMGB1; extracorporeal shock wave therapy; burn hypertrophic scar; hypertrophic scar-derived fibroblast; epithelial-mesenchymal transition; inhibitor of DNA binding protein; keloid; hypertrophic scar; scar biology; scar prevention; scar treatment; keloids; fibroproliferative disorder; HtrA1; inflammation; n/a