Reprint

COVID-19 Coagulopathy: Advances on Pathophysiology and Therapies

Edited by
April 2024
258 pages
  • ISBN978-3-7258-0964-6 (Hardback)
  • ISBN978-3-7258-0963-9 (PDF)

This book is a reprint of the Special Issue COVID-19 Coagulopathy: Advances on Pathophysiology and Therapies that was published in

Biology & Life Sciences
Chemistry & Materials Science
Medicine & Pharmacology
Summary

This Special Issue aims to dismantle the mechanisms and molecular pathways underlying COVID-19-induced coagulopathy, and present the current advances made possible by our colleagues’ research. The 15 published paper contributions encompassed molecular recruitment and binding, enzymatic dynamics and hemostasis disorders, as well as imagistic high-risk identifiers among the coronary disease population affected by the virus, the connection between psychiatric patients also affected and the thrombosis risk of their antipsychotic therapy, and complications in pediatric patients, along with those with primary and secondary immune disorders.

As underlined by the published papers, this Special Issue has brought important contributions in understanding the root mechanisms of SARS-CoV-2-induced coagulopathy, defining diagnostic and prognostic biomarkers, and establishing the role of anti-coagulant and anti-thrombotic therapies which are the most effective for this complex disease.

We are pleased to acknowledge that researchers from various fields came together in an effort to provide necessary data about the many forms and manifestations of the coagulations disorders challenging the medical community since the early stages of the pandemic. These data can represent a basis for further studies and a fertile ground for later developments.

Format
  • Hardback
License and Copyright
© 2024 by the authors; CC BY-NC-ND license
Keywords
acute pancreatitis; COVID-19; HLA-DR; monocytes; immune response; immunosuppression; COVID-19; thrombosis; coagulopathy; vasculopathy; inflammation; diabetes mellitus; COVID-19; post-acute COVID-19 syndrome; coagulopathy; fibrinolysis; thrombosis; COVID-19; vascular inflammation; fat attenuation index; plaque vulnerability; thrombosis; chronic coronary syndrome; pericoronary adipose tissue; pulmonary thrombosis; SARS-CoV-2; neonatal thrombosis; COVID-19 complications; COVID-19; bacterial infection; thrombosis; platelet activation; sCLEC-2; hypofibrinolytic state; COVID-19; SARS-CoV-2; disorder of immunity; cancer; COVID-19 infection; coagulopathy; endothelial dysfunction; platelet activation; citokine storm; anticoagulant therapy; COVID-19; SARS-CoV-2; RNase 1; kidney injury; biomarker; SARS-CoV-2 spike proteins; human lung endothelial cells; neutrophils; P-selectin; von willebrand factor; IL-6; citrullinated histone H3; neutrophils extracellular traps; TFPI; DTNB; thrombomodulin; COVID-19; viscoelastometry; hypercoagulable state; thrombosis; coagulopathy; venous thromboembolism; obesity; diabetes mellitus; cardiovascular diseases; stroke; COVID-19; inflammation; coagulopathy; diabetes mellitus; severity; SARS-CoV-2; spike protein; COVID-19; sialic acid; glycophorin A; hemagglutination; hemagglutinin esterase; antipsychotic medications; thrombosis; COVID-19; n/a