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Volume 13
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10.3390/cells13141167
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Correction to Cells 2022, 11(13), 2104.
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Correction

Correction: Ma et al. Extracellular Vesicles from BMSCs Prevent Glucocorticoid-Induced BMECs Injury by Regulating Autophagy via the PI3K/Akt/mTOR Pathway. Cells 2022, 11, 2104

by
Jinhui Ma
1,†,
Mengran Shen
2,†,
Debo Yue
1,
Weiguo Wang
1,
Fuqiang Gao
1,* and
Bailiang Wang
1,*
1
Department of Orthopaedic Surgery, Center for Osteonecrosis and Joint Preserving & Reconstruction, China-Japan Friendship Hospital, Beijing 100029, China
2
Department of Orthopaedic Surgery, Peking University China-Japan Friendship School of Clinical Medicine, Beijing 100029, China
*
Authors to whom correspondence should be addressed.
†
These authors contributed equally to this work.
Cells 2024, 13(14), 1167; https://doi.org/10.3390/cells13141167
Submission received: 15 April 2024 / Accepted: 24 May 2024 / Published: 9 July 2024
Browse Figure
Versions Notes
In the original publication [1], there was a mistake in Figure 5A as published. The panels for Model+EVs and Model+EVs+LY294002 were the same. The corrected Figure 5A appears below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Reference

  1. Ma, J.; Shen, M.; Yue, D.; Wang, W.; Gao, F.; Wang, B. Extracellular Vesicles from BMSCs Prevent Glucocorticoid-Induced BMECs Injury by Regulating Autophagy via the PI3K/Akt/mTOR Pathway. Cells 2022, 11, 2104. [Google Scholar] [CrossRef] [PubMed]
Cells 13 01167 g005
Figure 5. BMSC-EV-derived exosomes BMECs’ viability and protected BMECs against glucocorticoid-induced apoptosis. (A) Apoptosis was quantified by flow cytometry after staining with annexin V-FITC/PI. (B) Percentage of apoptotic cells in different groups. (C) CCK-8 was used to measure viability of BMECs in different groups. Each bar represents the mean ± SD of three independent experiments. ** p < 0.01.
Figure 5. BMSC-EV-derived exosomes BMECs’ viability and protected BMECs against glucocorticoid-induced apoptosis. (A) Apoptosis was quantified by flow cytometry after staining with annexin V-FITC/PI. (B) Percentage of apoptotic cells in different groups. (C) CCK-8 was used to measure viability of BMECs in different groups. Each bar represents the mean ± SD of three independent experiments. ** p < 0.01.
Cells 13 01167 g005
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MDPI and ACS Style

Ma, J.; Shen, M.; Yue, D.; Wang, W.; Gao, F.; Wang, B. Correction: Ma et al. Extracellular Vesicles from BMSCs Prevent Glucocorticoid-Induced BMECs Injury by Regulating Autophagy via the PI3K/Akt/mTOR Pathway. Cells 2022, 11, 2104. Cells 2024, 13, 1167. https://doi.org/10.3390/cells13141167

AMA Style

Ma J, Shen M, Yue D, Wang W, Gao F, Wang B. Correction: Ma et al. Extracellular Vesicles from BMSCs Prevent Glucocorticoid-Induced BMECs Injury by Regulating Autophagy via the PI3K/Akt/mTOR Pathway. Cells 2022, 11, 2104. Cells. 2024; 13(14):1167. https://doi.org/10.3390/cells13141167

Chicago/Turabian Style

Ma, Jinhui, Mengran Shen, Debo Yue, Weiguo Wang, Fuqiang Gao, and Bailiang Wang. 2024. "Correction: Ma et al. Extracellular Vesicles from BMSCs Prevent Glucocorticoid-Induced BMECs Injury by Regulating Autophagy via the PI3K/Akt/mTOR Pathway. Cells 2022, 11, 2104" Cells 13, no. 14: 1167. https://doi.org/10.3390/cells13141167

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