Correction: Dulski et al. An Organic–Inorganic Hybrid Nanocomposite as a Potential New Biological Agent. Nanomaterials 2020, 10, 2551

Error in Figure

In the original publication [1], there was a mistake in Figure 9, as published. The authors identified an error in the incorrect description of the y-axes’ titles. Specifically, in the lower panel of Figure 9b, the axis title “cell cycle stage [%]” was corrected to “cellular subpopulations [%]”. Similarly, in Figure 9c, the title “cell cycle stage [%]” was corrected to “ROS level [%]”. The corrected Figure 9 appears below. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.

Figure 9. Impact of the tested nanocomposites at a 30 mg/L concentration on regulating the cell cycle (a) and inducing apoptosis (b) in the PANC-1 cells. Effect of the tested Ag/SiO₂@CMC nanocomposite on the level of reactive oxygen species (ROS) in the PANC-1 cells. Data normalized to the untreated cells (control) (c). Impact of Ag/SiO₂@CMC on the expression of the p53, cyclin E1, and HO-1 proteins in the PANC-1 cells. The densitometric analysis of these proteins was normalized to GADPH (d). The results from all experiments are shown as the mean ± standard deviation (SD) of three independent measurements. Any statistical differences from the cell cycle, apoptosis, and immunoblotting experiments were analyzed using a one-way ANOVA with Bonferroni’s post-hoc test. Data from ROS measurements were analyzed using the Student’s t-test. Statistical significance: * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001 compared to the control group.

Figure 9. Impact of the tested nanocomposites at a 30 mg/L concentration on regulating the cell cycle (a) and inducing apoptosis (b) in the PANC-1 cells. Effect of the tested Ag/SiO₂@CMC nanocomposite on the level of reactive oxygen species (ROS) in the PANC-1 cells. Data normalized to the untreated cells (control) (c). Impact of Ag/SiO₂@CMC on the expression of the p53, cyclin E1, and HO-1 proteins in the PANC-1 cells. The densitometric analysis of these proteins was normalized to GADPH (d). The results from all experiments are shown as the mean ± standard deviation (SD) of three independent measurements. Any statistical differences from the cell cycle, apoptosis, and immunoblotting experiments were analyzed using a one-way ANOVA with Bonferroni’s post-hoc test. Data from ROS measurements were analyzed using the Student’s t-test. Statistical significance: * p < 0.05, ** p < 0.01, *** p < 0.001, **** p < 0.0001 compared to the control group.