Correction: Woodlief et al. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics 2021, 9, 100
Department of Pharmacology and Toxicology, Brody School of Medicine, East Carolina University, Greenville, NC 27858, USA
*
Author to whom correspondence should be addressed.
Toxics 2023, 11(8), 656; https://doi.org/10.3390/toxics11080656
Submission received: 14 June 2023
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Revised: 13 July 2023
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Accepted: 18 July 2023
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Published: 28 July 2023
(This article belongs to the Section Emerging Contaminants)
Error in Figure/Table
Error in figure x-axis (Figure 1A: Hepatic peroxisome proliferation). In the original publication [1] there was a mistake in Figure 1A as published. The x-axis was supposed to be labeled with the following doses: 0, 0.00025, 0.025, or 2.5 PFOA. The authors state that the scientific conclusions are unaffected. This correction was approved by the Academic Editor. The original publication has also been updated.
Reference
- Woodlief, T.; Vance, S.; Hu, Q.; DeWitt, J. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics 2021, 9, 100. [Google Scholar] [CrossRef] [PubMed]
Figure 1.
Hepatic peroxisome proliferation (percent change from 0 mg/kg control) of male and female C57BL/6 mice orally exposed to (A): PFMOAA, (B): PFMOPrA, or (C): PFMOBA for 30 days. Acyl-CoA oxidase activity was measured in livers that had been collected from animals one day after exposure ended. n = 4–6/dose for PFMOAA, PFMOPrA, PFMOBA, and PFOA-positive control (note that the PFOA-positive control was included from animals evaluated in a separate PFAS study). No error bars are present due to how the data were calculated. Abbreviations: perfluoro-2-methoxyacetic acid (PFMOAA), perfluoro-2-methoxypropanoic acid (PFMOPrA), perfluoro-4-methoxybutanioc acid (PFMOBA), and perfluorooctanoic acid (PFOA). * p < 0.05 from same-sex control group.
Figure 1.
Hepatic peroxisome proliferation (percent change from 0 mg/kg control) of male and female C57BL/6 mice orally exposed to (A): PFMOAA, (B): PFMOPrA, or (C): PFMOBA for 30 days. Acyl-CoA oxidase activity was measured in livers that had been collected from animals one day after exposure ended. n = 4–6/dose for PFMOAA, PFMOPrA, PFMOBA, and PFOA-positive control (note that the PFOA-positive control was included from animals evaluated in a separate PFAS study). No error bars are present due to how the data were calculated. Abbreviations: perfluoro-2-methoxyacetic acid (PFMOAA), perfluoro-2-methoxypropanoic acid (PFMOPrA), perfluoro-4-methoxybutanioc acid (PFMOBA), and perfluorooctanoic acid (PFOA). * p < 0.05 from same-sex control group.
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MDPI and ACS Style
Woodlief, T.; Vance, S.; Hu, Q.; DeWitt, J. Correction: Woodlief et al. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics 2021, 9, 100. Toxics 2023, 11, 656. https://doi.org/10.3390/toxics11080656
AMA Style
Woodlief T, Vance S, Hu Q, DeWitt J. Correction: Woodlief et al. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics 2021, 9, 100. Toxics. 2023; 11(8):656. https://doi.org/10.3390/toxics11080656
Chicago/Turabian StyleWoodlief, Tracey, Samuel Vance, Qing Hu, and Jamie DeWitt. 2023. "Correction: Woodlief et al. Immunotoxicity of Per- and Polyfluoroalkyl Substances: Insights into Short-Chain PFAS Exposure. Toxics 2021, 9, 100" Toxics 11, no. 8: 656. https://doi.org/10.3390/toxics11080656
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