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Xia, J.; et al., Arsenic Trioxide Inhibits Cell Growth and Induces Apoptosis through Inactivation of Notch Signaling Pathway in Breast Cancer. Int. J. Mol. Sci. 2012, 13, 9627–9641

1
Department of Biochemistry and Molecular Biology, Bengbu Medical College, Bengbu 233030, China
2
Laboratory Medicine, Taixing People's Hospital, Taizhou 225400, China
3
Research Center of Clinical Laboratory Science, Bengbu Medical College, Bengbu 233030, China
4
Department of Pathology and Oncology, Karmanos Cancer Institute, Wayne State University, Detroit, MI 48201, USA
5
University of Mississippi Cancer Institute, 2500 N State St., Jackson, MS 39216, USA
6
Department of Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, 330 Brookline Avenue, Boston, MA 02215, USA
*
Author to whom correspondence should be addressed.
These authors contributed equally to this work.
Int. J. Mol. Sci. 2014, 15(8), 14907-14908; https://doi.org/10.3390/ijms150814907
Submission received: 25 July 2014 / Accepted: 11 August 2014 / Published: 22 August 2014
The authors wish to change Figure 5D of the paper published in IJMS [1]. In Figure 5D, the bands for NF-κB and Bcl-2 are similar with Notch-1 bands. The authors have carefully checked the original files and found that it is an inadvertent mistake in the published version of Figure 5D. Figure 5 is revised as follows. The authors would like to apologize for any inconvenience caused to the readers by these changes.
Figure 5. The efficacy of transfection by Notch-1 siRNA and Notch-1 cDNA in SKBR-3 cells. A-D: The expression of Notch-1 was detected by RT-PCR and Western blotting, respectively, to check the Notch-1 siRNA transfection efficacy; E: The expression of Notch-1 was detected by Western blotting for assessing the Notch-1 cDNA plasmid transfection efficacy.
Figure 5. The efficacy of transfection by Notch-1 siRNA and Notch-1 cDNA in SKBR-3 cells. A-D: The expression of Notch-1 was detected by RT-PCR and Western blotting, respectively, to check the Notch-1 siRNA transfection efficacy; E: The expression of Notch-1 was detected by Western blotting for assessing the Notch-1 cDNA plasmid transfection efficacy.
Ijms 15 14907 g005

Reference

  1. Xia, J.; Li, Y.; Yang, Q.; Mei, C.; Chen, Z.; Bao, B.; Ahmad, A.; Miele, L.; Sarkar, F.; Wang, Z. Arsenic trioxide inhibits cell growth and induces apoptosis through inactivation of Notch signaling pathway in breast cancer. Int. J. Mol. Sci. 2012, 13, 9627–9641. [Google Scholar] [CrossRef]

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MDPI and ACS Style

Xia, J.; Li, Y.; Yang, Q.; Mei, C.; Chen, Z.; Bao, B.; Ahmad, A.; Miele, L.; Sarkar, F.H.; Wang, Z. Xia, J.; et al., Arsenic Trioxide Inhibits Cell Growth and Induces Apoptosis through Inactivation of Notch Signaling Pathway in Breast Cancer. Int. J. Mol. Sci. 2012, 13, 9627–9641. Int. J. Mol. Sci. 2014, 15, 14907-14908. https://doi.org/10.3390/ijms150814907

AMA Style

Xia J, Li Y, Yang Q, Mei C, Chen Z, Bao B, Ahmad A, Miele L, Sarkar FH, Wang Z. Xia, J.; et al., Arsenic Trioxide Inhibits Cell Growth and Induces Apoptosis through Inactivation of Notch Signaling Pathway in Breast Cancer. Int. J. Mol. Sci. 2012, 13, 9627–9641. International Journal of Molecular Sciences. 2014; 15(8):14907-14908. https://doi.org/10.3390/ijms150814907

Chicago/Turabian Style

Xia, Jun, Youjian Li, Qingling Yang, Chuanzhong Mei, Zhiwen Chen, Bin Bao, Aamir Ahmad, Lucio Miele, Fazlul H. Sarkar, and Zhiwei Wang. 2014. "Xia, J.; et al., Arsenic Trioxide Inhibits Cell Growth and Induces Apoptosis through Inactivation of Notch Signaling Pathway in Breast Cancer. Int. J. Mol. Sci. 2012, 13, 9627–9641" International Journal of Molecular Sciences 15, no. 8: 14907-14908. https://doi.org/10.3390/ijms150814907

APA Style

Xia, J., Li, Y., Yang, Q., Mei, C., Chen, Z., Bao, B., Ahmad, A., Miele, L., Sarkar, F. H., & Wang, Z. (2014). Xia, J.; et al., Arsenic Trioxide Inhibits Cell Growth and Induces Apoptosis through Inactivation of Notch Signaling Pathway in Breast Cancer. Int. J. Mol. Sci. 2012, 13, 9627–9641. International Journal of Molecular Sciences, 15(8), 14907-14908. https://doi.org/10.3390/ijms150814907

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