Bette, M.; Cors, E.; Kresse, C.; Schütz, B.
Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes. Int. J. Mol. Sci. 2022, 23, 3403.
https://doi.org/10.3390/ijms23063403
AMA Style
Bette M, Cors E, Kresse C, Schütz B.
Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes. International Journal of Molecular Sciences. 2022; 23(6):3403.
https://doi.org/10.3390/ijms23063403
Chicago/Turabian Style
Bette, Michael, Eileen Cors, Carolin Kresse, and Burkhard Schütz.
2022. "Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes" International Journal of Molecular Sciences 23, no. 6: 3403.
https://doi.org/10.3390/ijms23063403
APA Style
Bette, M., Cors, E., Kresse, C., & Schütz, B.
(2022). Therapeutic Treatment of Superoxide Dismutase 1 (G93A) Amyotrophic Lateral Sclerosis Model Mice with Medical Ozone Decelerates Trigeminal Motor Neuron Degeneration, Attenuates Microglial Proliferation, and Preserves Monocyte Levels in Mesenteric Lymph Nodes. International Journal of Molecular Sciences, 23(6), 3403.
https://doi.org/10.3390/ijms23063403
