The Plasticity of Th17 Cells in the Pathogenesis of Rheumatoid Arthritis
Abstract
:1. Introduction
2. Plasticity of Th17
3. Th17-Derived Th1 Cells (CD161+ Th1 Cells, Nonclassic Th1 Cells)
4. Th17-Producing IFNγ (IFNγ+ Th17)
5. Possible Implications for Novel Therapies
6. Conclusions
Author Contributions
Conflicts of Interest
References
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Kotake, S.; Yago, T.; Kobashigawa, T.; Nanke, Y. The Plasticity of Th17 Cells in the Pathogenesis of Rheumatoid Arthritis. J. Clin. Med. 2017, 6, 67. https://doi.org/10.3390/jcm6070067
Kotake S, Yago T, Kobashigawa T, Nanke Y. The Plasticity of Th17 Cells in the Pathogenesis of Rheumatoid Arthritis. Journal of Clinical Medicine. 2017; 6(7):67. https://doi.org/10.3390/jcm6070067
Chicago/Turabian StyleKotake, Shigeru, Toru Yago, Tsuyoshi Kobashigawa, and Yuki Nanke. 2017. "The Plasticity of Th17 Cells in the Pathogenesis of Rheumatoid Arthritis" Journal of Clinical Medicine 6, no. 7: 67. https://doi.org/10.3390/jcm6070067
APA StyleKotake, S., Yago, T., Kobashigawa, T., & Nanke, Y. (2017). The Plasticity of Th17 Cells in the Pathogenesis of Rheumatoid Arthritis. Journal of Clinical Medicine, 6(7), 67. https://doi.org/10.3390/jcm6070067