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1 November 2022

Selective Biomarkers of Oxidative Stress in Ischemic Stroke †

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1
Department of Medical Biology and Biochemistry, Faculty of Medicine, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Toruń, Karłowicza 24, 85-092 Bydgoszcz, Poland
2
Department of Cardiology and Clinical Pharmacology, Faculty of Health Sciences, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Toruń, Ujejskiego 75, 85-168 Bydgoszcz, Poland
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Division of Biochemistry and Biogerontology, Department of Geriatrics, Faculty of Health Sciences, Collegium Medicum in Bydgoszcz, Nicolaus Copernicus University in Toruń, Dębowa 3, 85-626 Bydgoszcz, Poland
4
Centre of Medical Simulations, Medical University of Gdańsk, Dębowa 25, 80-204 Gdańsk, Poland

Abstract

Stroke is a serious health problem all over the world and the second most common cause of death and permanent disability in people, after heart attacks. Oxidative stress plays an important role in the pathogenesis of acute ischemic stroke (AIS). The aim of our study was to evaluate the temporal profile of the melatonin metabolite 6-hydroxymelatonin sulphate (6-SM) in the urine and carbonyl groups in the serum of patients with acute ischemic stroke treated with intravenous thrombolysis. There were statistically significant differences between the values of 6-SM and carbonyls compared to the control group. Statistical differences were also found in the concentrations of the examined parameters depending on the type of stroke. The correlations between the concentrations of 6-SM and the carbonyl groups measured in various time intervals and between the concentration of biomarkers and mortality was also evaluated. The results indicate increased oxidative stress and the intense increase in the concentration of carbonyl groups in patients with AIS, which could be potential markers of protein damage in thrombolytic patients. Melatonin supplementation in AIS patients can also be considered, as it can effectively prevent both behavioral and neurophysiological defects caused by cerebral hypoxia and ischemia.

Supplementary Materials

The following are available online at https://www.mdpi.com/article/10.3390/ECMC2022-13156/s1.

Author Contributions

Conceptualization, H.P.; methodology, H.P., M.K., R.K. and M.P.; validation, H.P., R.K.; investigation, H.P., M.K., M.P., M.G.; resources, H.P., E.G. and G.K.; writing—original draft preparation, H.P. and R.K.; writing—review and editing, H.P., R.K., A.K., G.G., A.W. and G.K.; visualization, H.P. and M.P.; supervision, H.P.; project administration, H.P.; funding acquisition, H.P., A.W. and R.K.; All authors have read and agreed to the published version of the manuscript.

Funding

This work has been supported by Nicolaus Copernicus University as part of the Initiative of excellence competition—Research University—“Debuts” (10/2022) and Research Project, Collegium Medicum in 2022.

Institutional Review Board Statement

The study was conducted in accordance with the Declaration of Helsinki, and approved by the local Ethics Committee (No KB 637/2016).

Data Availability Statement

Data available from the authors.

Conflicts of Interest

The authors declare no conflict of interest.
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