Autophagy Contribution to Cancer Therapy Resistance
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Autophagy".
Deadline for manuscript submissions: closed (15 July 2024) | Viewed by 785
Special Issue Editors
Interests: autophagy; signaling; metabolism; acute myeloid leukemia
Special Issues, Collections and Topics in MDPI journals
Interests: host autophagy; metabolism; immune response; acute myeloid leukemia
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Unfortunately, for many types of cancer, therapy resistance is common and accounts for most cases of treatment failure. Therefore, the identification of new targets and resistance mechanisms is urgently needed to improve clinical outcomes. The well-conserved catabolic process, autophagy, has previously been implicated in the initiation, progression, and therapeutic resistance of numerous tumors. This survival mechanism, characterized by the lysosomal degradation of cytoplasmic content and damaged organelles, ensures energy and cellular homeostasis. In addition, especially through its role in metabolism regulation, tumor and microenvironmental autophagy have been increasingly associated with oncogenesis. However, studies to better understand its role and identify through which molecular and cellular mechanisms autophagy regulates response to therapy, especially in vivo, are still necessary.
This Special Issue will investigate, in vitro and in vivo, the contribution of autophagy to cancer resistance and relapse, depending on tumor type and the treatments used, in order to understand whether autophagy modulation could be an interesting strategy to improve therapy efficacy.
Dr. Carine Joffre
Dr. Laura Poillet
Guest Editors
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Keywords
- autophagy
- cancers
- therapy resistance
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