Mechanisms of Neuronal Death during Neurodegenerative Disease and the Fundamentals of Synaptic Plasticity
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Neurobiology".
Deadline for manuscript submissions: 31 December 2024 | Viewed by 114
Special Issue Editors
Interests: inflammation; neurodegeneration; Alzheimer’s disease; Parkinson’s disease
Interests: neurodegenerative diseases; Alzheimer’s disease; Parkinson’s disease; Huntington’s disease
Special Issue Information
Dear Colleagues,
Neurodegeneration is the process that is characterized by neuronal loss eventually leading to cognitive and motor dysfunctions. The brain is especially consumed by changes associated with aging, resulting in neurodegeneration and different neurodegenerative diseases. The mechanisms related to neurodegeneration are broad and encompass neuroinflammation, mitochondrial dysfunction, oxidative stress, altered synaptic plasticity, and changes in protein homeostasis, among others. The precise mechanisms related to neurodegenerative diseases are not well understood; therefore, therapeutic approaches are limited in their efficacy and validity. We know that the multitude of neurodegenerative diseases have similarities and differences that make them similar targets but also unique in their approach to treatments. Thus, an in-depth understanding of the specifically related mechanisms associated with neurodegenerative diseases may contribute to the identification of new targets for the treatment of neurodegenerative diseases.
The purpose of this Special Issue is to present the latest research on neurodegeneration and the potential mechanisms or therapeutic approaches to treating the disorders. Both original research and comprehensive reviews are welcome.
Prof. Dr. Mark Kindy
Prof. Dr. Santosh D’Mello
Prof. Dr. Kumar Sambamurti
Guest Editors
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Keywords
- neurodegeneration
- Alzheimer’s disease
- Parkinson’s disease
- amyotrophic lateral sclerosis
- in-flammation
- oxidative stress
- synaptic plasticity
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