The Oxidative Stress Dilemma: How Cellular Imbalance Leads to Disease?
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: 30 November 2024 | Viewed by 149
Special Issue Editor
Interests: the role of the microbiome in the development and therapeutic effect of cancer; the relationship and mechanism of microbiome-derived substances with cell death, autophagy; mitophagy
Special Issue Information
Dear Colleagues,
Mitochondria are the primary site for ROS production during oxidative phosphorylation. Additionally, increased ROS production due to exposure to environmental factors such as pollutants, radiation, and chemicals is associated with the development of various diseases. While physiological levels of ROS act as signalling molecules to maintain healthy body functions, elevated levels of ROS can abnormally affect organ and system homeostasis, ultimately promoting disease onset and progression. Additionally, depending on the body's redox environment and its concentration, many xenobiotics can abnormally interact with the organ's redox regulatory machinery, ultimately facilitating cell and tissue damage due to oxidative stress. Cancer, cardiovascular and metabolic diseases, inflammatory diseases, autoimmune diseases, and chronic degenerative diseases all commonly exhibit imbalanced redox homeostasis due to increased ROS production or defects in the body's antioxidant responses.
This special issue will cover recent discoveries in the field of identifying reliable oxidative stress biomarkers to improve early diagnosis and treatment monitoring of oxidative stress-related diseases.
Dr. Hyunju Kim
Guest Editor
Manuscript Submission Information
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Keywords
- oxidative stress
- biomarker
- pharmacological advance
- lifestyle and dietary intervention
- autophagy (mitophagy)
- environmental factors
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