Molecular and Cellular Mechanisms of Chronic Obstructive Pulmonary Disease (COPD)
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cellular Pathology".
Deadline for manuscript submissions: closed (25 July 2024) | Viewed by 3605
Special Issue Editor
Interests: chronic lung disease; COPD; house hold air pollution (HAP); LIC; LMIC; electronic cigarettes; heated tobacco products
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Chronic obstructive pulmonary disease (COPD) is the third leading cause of death worldwide. Tobacco smoking, exposure to biomass smoke, air pollution, and occupation exposures are regarded as some of the major causes of COPD. Chronic bronchitis and emphysema are commonly associated with COPD. Classical inflammatory reactions and oxidative stress are considered as the major molecular pathways upstream of COPD pathogenesis. However, current knowledge points towards the involvement of much broader mechanisms. In order to discover new targets, identify robust biomarkers, and develop novel therapies, there is a need to develop a deeper understanding of the molecular and cellular mechanisms.
This Special Issue aims to summarize the current knowledge and advances in the COPD area of research, including studies that cover diagnostic tools, basic molecular mechanisms, pharmacotherapy, toxicological risk assessments, genomics, and computational modeling of diseased lung. We invite you to submit original research articles and comprehensive review articles that cover the newest findings in these fields.
Dr. Koustav Ganguly
Guest Editor
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Keywords
- chronic obstructive pulmonary disease
- COPD
- smoke
- pharmacotherapy
- toxicological
- genomics
- computational modeling
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