Molecular and Cellular Mechanisms of Idiopathic Pulmonary Fibrosis and Interstitial Lung Diseases
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Pathology, Diagnostics, and Therapeutics".
Deadline for manuscript submissions: closed (20 May 2023) | Viewed by 117487
Special Issue Editor
Interests: lung pathology; interstitial lung disease; idiopathic pulmonary fibrosis; molecular pathology
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
It is time to shed light on the molecular and cellular mechanisms of lung fibrosis!
Lungs are exposed to many insults with every breath we take, showing an extraordinary ability to repair damaged cells and tissues. A complex interplay between genetic background and the environment drives the response of lung tissues to acquired damage, and lung fibrosis is, in most cases, the final result of a maladaptive response to this complex interaction. Lung fibrosis causes a decline in pulmonary function, with patients suffering a worse quality of life and poor prognosis with early mortality.
Idiopathic pulmonary fibrosis (IPF) is a prototype of chronic, progressive interstitial lung disease (ILD), in which progressive lung scarring involves the lungs interstitium; although thought to be rare, its incidence has recently increased in the Western world and is similar to some types of cancer. At the present time, therapeutical options for treating IPF and other ILD are scarce and show little benefit.
This Special Issue is awaiting the submission of scientific research articles and reviews that describe the molecular and cellular mechanisms driving the progression of fibrosing lung diseases, with a particular focus on IPF, for a better understanding of their pathogenesis and the development of diagnostic markers, as well as the discovery of molecular targets suitable for tailored therapies.
Dr. Massimiliano Mancini
Guest Editor
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Keywords
- lung fibrosis
- idiopathic pulmonary fibrosis
- interstitial lung disease
- molecular pathways
- cellular aging and senescence
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