Redox Regulation in a Tumor Microenvironment
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".
Deadline for manuscript submissions: closed (31 May 2022) | Viewed by 461
Special Issue Editors
Interests: nutritional and oxidative stress in cancers
Special Issues, Collections and Topics in MDPI journals
2. Centre Scientifique de Monaco (CSM), 8 Quai Antoine Ier MC 98000 Monaco, Monaco
Interests: Hypoxic signaling and cancer metabolism; Acid, nutritional and oxidative stress in cancers
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
The tumor microenvironment (TME) has been recognized as one of the major players in tumor progression, dissemination and chemoresistance. The term TME refers to a complex and dynamic system composed of many different cell types, such as fibroblasts, immune cells and endothelial cells, which are immersed in the mesh of the extracellular matrix (ECM). The reciprocal interaction between cancer and neighbouring cells, as well as the components of ECM, can be studied from various aspects of cancer biology, but the overarching component of all these aspects is their redox nature. This is, at the same time, one of the most puzzling questions in cancer biology considering that the line between pro- and anti-tumorigenic effects of reactive oxygen species (ROS) is still poorly defined. Thus, further comprehensive fundamental and pre-clinical research is necessary in order to gain a better understanding of the redox nature of cancer initiation and progression, to exploit its vulnerability points and to anticipate and prevent potential anti-cancer therapeutic-resistant mechanisms.
This Special Issue, “Redox Regulation in a Tumor Microenvironment”, of the journal Antioxidants welcomes both review and original articles.
Dr. Milica VUCETIC
Dr. Jacques Pouyssegur
Guest Editors
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Keywords
- Redox regulation
- Cancer
- Tumor Microenvironment
- Hypoxia
- Metabolism
- Nutrient Availability
- Cell Death
- Ferroptosis
- Cancer-Associated Fibroblasts
- Immune Cells
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