Molecular Mechanisms of Nerve Injury and Neuropathic Pain
A special issue of Biomedicines (ISSN 2227-9059). This special issue belongs to the section "Neurobiology and Clinical Neuroscience".
Deadline for manuscript submissions: closed (31 July 2022) | Viewed by 2089
Special Issue Editors
Interests: brain; neuropathic pain; astrocyte; insular cortex; MEMRI
Special Issue Information
Dear Colleague,
Neuropathic pain is caused by lesions or diseases of the somatosensory system and is a major public health issue in the world. It is characterized by increased responses to nociceptive stimuli (hyperalgesia), unpleasant and abnormal sensations (dysesthesia), and pain in response to light tactile stimuli (allodynia). Studies show that neuropathic pain involves a series of pathophysiologic events when the nerve is damaged, including neuronal hyperexcitability, changes in perineuronal homeostasis, and neurogenic inflammation. Although the treatment for neuropathic pain remains a great challenge, progress has been made in identifying key molecules and their roles in pain modulation and processing.
The purpose of this topic is to gather original research articles, reviews, and perspectives to advance our understanding of novel neuropathic pain-related molecules and signaling pathways that can be used as therapeutic targets for the treatment of pain. Subtopics of this topic include but are not limited to:
- Novel molecules and their roles in neuropathic pain modulation and processing;
- Transcriptional factors, membrane proteins, and ion channels, which are implicated in the pathological changes in somatosensory system;
- Identification of potential therapeutic targets and pathological mechanisms of neuropathic pain.
Neuropathic pain includes cancer pain, diabetic neuropathic pain, chemotherapy-induced pain, chronic postoperative pain, postherpetic neuralgia, etc. Both animal models and human studies are welcome.
Dr. Myeounghoon Cha
Dr. Junesun Kim
Guest Editors
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Keywords
- neuropathic pain
- hyperalgesia
- allodynia
- dysesthesia
- doral root ganglia
- spinal cord
- brain
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