Mitochondria and Central Nervous System Disorders: 3rd Edition
A special issue of Biomolecules (ISSN 2218-273X).
Deadline for manuscript submissions: 31 January 2025 | Viewed by 105
Special Issue Editors
2. Cologne Excellence Cluster on Cellular Stress Responses in Aging-Associated Diseases, University of Cologne, Cologne, Germany
Interests: mitochondria; translation; autophagy; neurons
Special Issues, Collections and Topics in MDPI journals
Interests: mitochondria; metabolism; transcriptomics; epigenetics
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Following the success of the previous Special Issues, we are pleased to announce the launch of a third edition, “Mitochondria and Central Nervous System Disorders: 3rd Edition”.
In the past several decades, a mitochondria-centric vision has developed in the fields of cell, organ, and organismal physiology, demonstrating exponential growth. This is likely due to the number of findings highlighting the contribution of these organelles to cell/tissue bioenergetics, death programmes, and metabolism. Dysfunctional mitochondria or dysfunctional mitochondria dynamics (a term that includes processes dictating the morphology of these organelles, their subcellular distribution/transport, or their interaction with other organelles, consequently influencing their function) have been linked to many pathological conditions, widespread among the entire human body. However, these alterations appear to more strongly affect the highly specialized and delicate cells of the central nervous system (CNS), contributing to the onset of a variety of diseases, ranging from rare childhood disorders (e.g., Leigh syndrome or mitochondrial encephalopathy with lactic acidosis and stroke-like episodes) to more common age-related neurodegenerative conditions (e.g., dementia, Alzheimer’s disease, and Parkinson’s disease).
This Special Issue is designed to emphasize the link between the (dys)function of mitochondria and CNS disorders, likely highlighting common or discrepant mechanisms underlying them. In this regard, we would like to invite review articles that address the above-mentioned topics or original research papers providing new evidence on the mitochondria–CNS pathological connection.
We look forward to reading your contributions.
Dr. Marta Zaninello
Dr. Camilla Bean
Guest Editors
Manuscript Submission Information
Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.
Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Biomolecules is an international peer-reviewed open access monthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- mitochondria
- metabolism
- neurodegeneration
- neuroinflammation
- ageing
- central nervous system
