Epstein-Barr Virus Disease Mechanisms and Stress Responses: 2nd Edition

A special issue of Biomolecules (ISSN 2218-273X). This special issue belongs to the section "Molecular Medicine".

Deadline for manuscript submissions: 30 November 2024 | Viewed by 241

Special Issue Editor


E-Mail Website
Guest Editor
1. Department of Cancer Biology and Genetics, The Ohio State University College of Medicine, Columbus, OH 43210, USA
2. Institute for Behavioral Medicine Research, The Ohio State University College of Medicine, Columbus, OH 43210, USA
Interests: herpesviruses lytic replication; innate and adaptive immunity; therapeutic interventions
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Special Issue Information

Dear Colleagues,

Following a very successful first publication, we are pleased to announce the launch of a second edition of a Special Issue entitled “Epstein-Barr Virus Disease Mechanisms and Stress Responses II”.

The virome of humans is complex and is composed of numerous virus families, including several members of the Herpesviridae. One member, the Epstein-Barr virus (EBV), a gamma herpesvirus, establishes persistent infections in more than 90% of adults worldwide. EBV is the etiological agent of several human cancers, and it has also been associated with an increased risk for developing several autoimmune diseases. However, the mechanisms through which EBV may contribute to these autoimmune diseases are unknown. Likewise, little is known concerning the role that EBV may have in modulating the host immune system in asymptomatic “healthy” individuals, and how stress may alter these responses during the aging process.

This Special Issue of Biomolecules is devoted to exploring the role of how molecules produced during the reactivation/partial reactivation of EBV may contribute to modifying the host’s immune system in immunocompetent individuals, and how stress may affect these processes. The main goal is to compile articles that describe recent progress in this area.

Prof. Dr. Marshall Vance Williams
Guest Editor

Manuscript Submission Information

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Keywords

  • Epstein–Barr virus (EBV)
  • reactivation
  • immune modulation
  • stress
  • aging

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