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The TNF Receptor Superfamily in Health and Disease
This special issue belongs to the section “Cell Signaling“.
Special Issue Information
Dear Colleagues,
This Special Issue is meant to collate articles that highlight our current knowledge of the important role that TNF receptor superfamily members play in essential functions such as immune regulation, cell proliferation, cell death, and morphogenesis. Contributions can focus on the signaling mechanism of any TNF receptor superfamily members or downstream signaling partners, their role in health and disease, and therapeutic targeting.
Prof. Dr. Denise Faustman
Guest Editor
Manuscript Submission Information
Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 250 words) can be sent to the Editorial Office for assessment.
Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Cells is an international peer-reviewed open access semimonthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- TNF receptor superfamily
- TNF signaling
- Apoptosis
- Cell proliferation
- Cancer
- Autoimmunity
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