Dopamine Signaling: From Synapses to Behavior
A special issue of Cells (ISSN 2073-4409). This special issue belongs to the section "Cells of the Nervous System".
Deadline for manuscript submissions: closed (10 March 2022) | Viewed by 35996
Special Issue Editor
Interests: basal ganglia; cellular excitability; motor control; dopamine-dependent plasticity; Parkinson’s disease; synaptic transmission; dopaminergic neurons ; dopamine receptors
Special Issue Information
Dear Colleagues,
Dopamine (DA) is a catecholamine which is instrumental for learning and motivation. DA is synthesized and released by midbrain dopamine neurons which innervate motor and limbic circuits to regulate ongoing behaviors. DA acts on D1- and D2-like receptor families to dynamically modulate cellular excitability and synaptic plasticity in a circuit-specific manner, and to ultimately control motivated behaviors. Most recent studies have unraveled new mechanisms about DA signaling such as co-release of neurotransmitters by DA terminals and local control of DA release by receptors located on synaptic terminals independently of dopaminergic neuron firing activity. In addition, the use of genetically-encoded biosensors and subcellular approaches tends toward a re-evaluation of the anatomical organization of DA release sites and suggests a more rapid DA coding than originally described. These new findings profoundly alter our current knowledge of DA functions both in healthy and diseased conditions.
The aim of this Special Issue is to present articles that investigate molecular, synaptic, and circuit mechanisms underlying DA signaling in order to better understand how DA sculpts neuronal activity and DA-dependent behaviors.
Dr. Jérôme Baufreton
Guest Editor
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Keywords
- dopamine synthesis and release
- dopamine receptors signaling
- basal ganglia
- limbic circuits
- dopamine-dependent plasticity
- motor control
- learning
- movement disorders
- motivation
- schizophrenia
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