Exploring the Molecular Mechanisms of Chronic Kidney Disease
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Pathology, Diagnostics, and Therapeutics".
Deadline for manuscript submissions: 20 April 2025 | Viewed by 2007
Special Issue Editor
Interests: bone disease; mineral metabolism; renal disease; vascular calcification; calcium-sensing receptor; fibroblast growth factor 23; sclerostin; vitamin D; parathyroid hormone
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Chronic kidney disease (CKD) is a global challenge, currently affecting approximately 1 in 10 individuals, accounting for over 800 million people with CKD worldwide. Unfortunately, the prevalence of CKD is increasing, estimated to become the fifth leading cause of death by 2040, which underscores the significant social and economic impact of this devastating disease. Research efforts focus on the prevention of CKD progression and associated complications that usually lead to increased hospitalization and higher mortality risk. Understanding the distinct molecular mechanisms underlying CKD development is key for identifying new therapeutic approaches and achieving the optimization of patient care.
This Special Issue aims to compile cutting-edge research on the molecular features of renal diseases and CKD-associated disorders, including metabolic bone disease, anemia, cardiovascular disease, diabetes and hypertension. In addition, novel molecular actions of current treatments and emerging drugs, as well as future strategies for preventing complications associated with dialysis or renal transplantation are also included.
Dr. Juan Miguel Diaz-Tocados
Guest Editor
Manuscript Submission Information
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Keywords
- chronic kidney disease
- hereditary renal disease
- renal hypertension
- diabetic nephropathy
- secondary hyperparathyroidism
- cardiovascular disease
- renal osteodystrophy
- peritoneal dialysis
- hemodialysis
- anemia
- metabolic acidosis
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