Exercise Interventions to Inflammation and Oxidative Stress
A special issue of Life (ISSN 2075-1729). This special issue belongs to the section "Physiology and Pathology".
Deadline for manuscript submissions: closed (30 September 2023) | Viewed by 4083
Special Issue Editor
Special Issue Information
Dear Colleagues,
Oxidative stress (OS) is triggered by imbalances between antioxidant defenses and damage caused by reactive oxygen species. OS can cause injuries to a variety of cells as well as DNA and is involved in the mechanism of multiple diseases such as cancer, metabolic syndrome, cardiovascular disease (CVD), and neurodegenerative disorder. Inflammation is also an essential immune response, and high inflammation levels can be found in many chronic diseases: obesity is a classic example characterized by low-grade chronic inflammation. In recent years, various studies have reported that exercise training and regular physical activity achieved subclinical effects in improving imbalances between oxidant production and antioxidant capacity, as well as in reducing inflammation levels. By contrast, acute exercise may increase OS. For instance, exercise-induced OS can injure myocytes, triggering an inflammatory response and muscle atrophy, while also playing an important role in regenerating muscles injured by an inflammatory response. However, the mechanism underlying the contrasting aspects of exercise is not yet fully understood. This Special Issue contains studies based on recent evidence associated with exercise interventions pertaining to inflammation and OS using human and animal models. Environmental stresses such as particulate matter and hypoxic conditions are primary factors under consideration.
Dr. Hee-Tae Roh
Guest Editor
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Keywords
- physical activity
- acute exercise
- exercise training
- inflammation
- oxidative stress
- antioxidant
- chronic disease
- environmental stress
- brain health
- aging
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