Redox and Nitrosative Signaling and Stress
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "ROS, RNS and RSS".
Deadline for manuscript submissions: closed (30 September 2020) | Viewed by 38172
Special Issue Editor
Interests: cardiovascular system; physiological endothelial functions; coronary circulation; metabolism; contractility; myocardial protection; endothelial cells; vitamins
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Redox/nitrosative signaling levels intervene in many human physiological adaptations and in many pathophysiological processes, leading to diseases such as cancer, neurodegenerative disease, lung disease, cardiovascular diseases, and ischemia/reperfusion injury. Recently, redox approaches have also shown promise in enhancing the levels of tolerance to cardio-toxic anticancer drugs and protecting from or inhibiting the progression of these diseases. The main scope of the present Special Issue is to reach a broad audience of scientists working in the field of redox biomedicine. We encourage the submission of papers approaching the topic from different points of view and at different levels, from basic to translational research in several cardiovascular fields.
We invite you to submit your latest research findings or a review article to this Special Issue, which will bring together current research concerning redox and nitrosative signaling both in normal processes and diseased states. This research can include both in vitro and in vivo studies relating to any of the following topics, though not exclusively: regulation of antioxidant enzymes; redox dependent post-translational modifications of proteins; role of redox state in cell metabolism, cell cycle, epigenetic regulation, cellular stress, and disease. Studies on organ protection and organ toxicity are also welcome.
We look forward to your contribution.
Prof. Dr. Claudia Penna
Guest Editor
Manuscript Submission Information
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