Oxidative Stress in Human Toxicology
A special issue of Antioxidants (ISSN 2076-3921). This special issue belongs to the section "Health Outcomes of Antioxidants and Oxidative Stress".
Deadline for manuscript submissions: closed (30 October 2022) | Viewed by 40212
Special Issue Editor
Special Issue Information
Dear Colleagues,
This Special Issue will focus on exposure to foreign chemicals (xenobiotics) that cause oxidative stress.
Certain xenobiotics and/or their metabolites (e.g. quinones) can directly mediate formation of reactive oxygen species (ROS, e.g. peroxides) that may induce damage to biomolecules. Xenobiotics can also indirectly cause oxidative stress by affecting protective proteins (e.g. antioxidant enzymes, catalytic transition metal transporters or chelators), regulation of vitamins, antioxidants, metals, or else, disturbances that may be toxic. Experimental studies in humans and human relevant models including air-breathing mammals (e.g. rodents, dogs, sheep), extensive in vitro (e.g. human cell cultures) studies, but also studies on ‘effect biomarkers of oxidative stress’ (e.g. from lipid peroxidation or nucleic acid damage) often measured in blood or urine, are welcome. Review articles that describe new mechanisms/mode of actions/key events, e.g. full, or parts of Adverse Outcome Pathways (AOPs) related to toxicants and oxidative stress can also be submitted.
Dr. Tim Hofer
Guest Editor
Manuscript Submission Information
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Keywords
- carbonyl
- DNA damage
- lipid peroxidation
- mitochondria
- nucleic acid
- protein oxidation
- radical
- repair
- redox cycling
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