From Molecular Mechanisms to Therapies: Advances in Retinal Disease Research

A special issue of Biomolecules (ISSN 2218-273X). This special issue belongs to the section "Molecular Biology".

Deadline for manuscript submissions: 31 March 2026 | Viewed by 42

Special Issue Editors


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Guest Editor
European Brain Research Institute, 00161 Rome, Italy
Interests: retina; oxidative stress; neurodegenerative diseases
Special Issues, Collections and Topics in MDPI journals

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Guest Editor

Special Issue Information

Dear Colleagues,

We would like to invite you to contribute to this Special Issue by submitting review articles that provide the most advanced information about the molecular causes of retinal diseases, in addition to the most developed therapeutic approaches for the retinal pathologies.

The retina is a fundamental tissue of the eye that makes it possible to transform the electrochemical stimuli elicited from the light into images through transporting the signal to the visual cortex. During aging, the retina tends to degenerate in a large portion of the population, leading to the onset of different retinal diseases (RDs). These conditions can result in loss of retinal function, which progresses into blindness.

It has been estimated that worldwide, between 7 and 8 million people are blind due to retinal diseases (accounting for overlaps and primary cause classifications). Additionally, over 13 million people suffer from severe vision impairment (MSVI) due to these conditions. The main contributing conditions are diabetic retinopathy, AMD, glaucoma, and retinitis pigmentosa.

In this Special Issue, we would like to highlight the latest knowledge about the molecular (genetic, biochemical and epigenetic) causes leading to these retinal pathologies and the new potential therapeutic approaches.

Dr. Marco Feligioni
Dr. Maccarone Rita
Guest Editors

Manuscript Submission Information

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Keywords

  • retinal diseases
  • retinal pathologies
  • preclinical studies

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Published Papers

This special issue is now open for submission.
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