Muscle Atrophy and Hypertrophy: Mechanisms and Potential Therapies
A special issue of Biomolecules (ISSN 2218-273X). This special issue belongs to the section "Molecular Medicine".
Deadline for manuscript submissions: closed (31 October 2023) | Viewed by 10432
Special Issue Editors
Interests: p38α-MAPK signaling; skeletal muscle
Interests: skeletal muscle; tendon; aging; muscle denervation; muscle unloading; obesity; myokines
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Skeletal muscle is essential for locomotion, maintaining posture, respiration (diaphragm), and phonation (laryngeal muscles). In addition, skeletal muscle plays a critical role in the whole-body metabolism. Skeletal muscle atrophy caused by a variety of conditions severely impairs muscle function. Skeletal muscle hypertrophy is adaptation to exercise and some other physiological processes. Muscle hypertrophy can improve muscle structure and function. Despite numerous published studies, the mechanisms regulating skeletal muscle atrophy and hypertrophy are still not completely understood.
In this Special Issue, we invite manuscripts on recent advances in the understanding of processes regulating skeletal muscle atrophy associated with diseases, aging, and unloading and muscle hypertrophy in response to exercise and other physiological adaptations.
A new Special Issue will be published in the Open Access journal Biomolecules (ISSN 2218-273X; 5-Year IF 6.19): “Muscle Atrophy and Hypertrophy: Mechanisms and Potential Therapies”.
In this Special Issue, we invite authors to submit original research and review articles revealing mechanisms of skeletal muscle atrophy and hypertrophy, including but not limited to the following topics:
- Signaling mechanisms involved in skeletal muscle atrophy and hypertrophy;
- Mechanisms of unloading-induced changes in gene expression and signaling in skeletal muscle;
- Denervation-induced skeletal muscle atrophy;
- Aging-related skeletal muscle atrophy;
- Skeletal muscle atrophy caused by diseases (diabetes, Duchenne atrophy, etc.);
- Tenotomy-related skeletal muscle atrophy;
- Exercise-induced muscle hypertrophy;
- Hormones and growth factors-induced skeletal muscle atrophy and hypertrophy;
- Role of satellite cells in muscle hypertrophy;
- Regulatory mechanisms of muscle protein synthesis involved in atrophy and hypertrophy;
- Therapies to counteract muscle atrophy.
Dr. Tatiana L. Nemirovskaya
Dr. Tatiana Kostrominova
Guest Editors
Manuscript Submission Information
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Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Biomolecules is an international peer-reviewed open access monthly journal published by MDPI.
Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2700 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.
Keywords
- skeletal muscle
- atrophy
- unloading
- denervation
- aging
- tenotomy
- gene expression
- signaling pathways
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