Cell Stress, Metabolism, Tumor Microenvironment in Ovarian Cancer
A special issue of Cancers (ISSN 2072-6694). This special issue belongs to the section "Tumor Microenvironment".
Deadline for manuscript submissions: closed (31 October 2022) | Viewed by 9109
Special Issue Editors
Interests: ovarian cancer; lung cancer; deubiquitination; non-coding RNAs; preclinical models of cancer; novel therapeutics
Interests: ovarian cancer, platelet biology, complement system, immune response, immunotherapy, novel therapeutics; interaction between platelet and complement system; cancer microenvironment
Special Issue Information
Dear Colleagues,
Ovarian cancer is the most lethal malignancy of the female reproductive system. Understanding of ovarian cancer initiation, progression, and metastasis is crucial to overcoming the disease. Genome, transcriptome, and proteome studies have shown that the ovarian tumor microenvironment (TME) plays a vital role in the tumorigenesis and progression of ovarian cancer. Ovarian TME, including malignant cells, contains the extracellular matrix, chemokines, cytokines, integrins, matrix metalloproteinases and other secreted molecules, stromal cells, cancer stem cells, pericytes, cancer-associated fibroblasts, endothelial cells, and immune cells. An advanced understanding of the interaction between ovarian cancer cells and the tumor microenvironment may present novel therapeutic targets for ovarian cancer. This Special Issue is aimed at highlighting recent studies on how various ovarian cancer cell intrinsic stresses, including DNA replication stress, DNA damage, genomic, epigenomic, transcriptomic changes, and metabolic reprograming, impact the tumor microenvironment and ovarian cancer progression and treatment. This issue also discusses the various therapies targeting ovarian cancer and the tumor microenvironment.
Dr. Cecil Han
Dr. Min Soon Cho
Guest Editors
Manuscript Submission Information
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Keywords
- ovarian cancer
- cell stress
- tumor microenvironment
- tumor metabolism
- metastasis
- immune response
- innate immunity
- adaptive immunity
- immunotherapy
- therapeutics
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