Cell Apoptosis, 3rd Edition
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Biology".
Deadline for manuscript submissions: 30 November 2024 | Viewed by 1687
Special Issue Editor
Interests: apoptosis; caspase; cell cycle; cell proliferation; cytotoxicity; in vitro antiproliferative activity; mitogen-activated protein kinases; reactive oxygen species; structure–activity relationship
Special Issues, Collections and Topics in MDPI journals
Special Issue Information
Dear Colleagues,
Apoptosis is a form of regulated cell death that plays a major role in an array of physiological and pathophysiological conditions. The execution of apoptosis is triggered by distinct and interconnected signaling pathways. Together with sustained proliferative signaling, the resistance to apoptosis is recognized as one of the cellular hallmarks of cancer. Exponential progress in our understanding of the regulation of death and survival signaling cascades has recently been achieved. This includes the executioners of apoptosis, B-cell lymphoma 2 family proteins, and oxidative stress. New advances in this field will allow for the identification of new modulators of the pathways in cell death and survival. This knowledge may contribute to the preparation of therapeutic strategies of interest in medicine. It includes the ability to potentiate the cytotoxicity against malignant cells and has potential application in other metabolic or immunological diseases. This Special Issue aims to improve our understanding of the molecular mechanisms involved in apoptosis. Contributions may relate to the events triggered by different physical and chemical agents, including naturally occurring and synthetic compounds, and/or by physiological and pathophysiological conditions.
More published papers can be found in the closed Special Issue “Cell Apoptosis” and “Cell Apoptosis 2.0”.
Prof. Dr. Francisco Estevez
Guest Editor
Manuscript Submission Information
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Keywords
- apoptosis
- caspase
- cell cycle
- cytotoxicity
- reactive oxygen species
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