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Sudden Cardiac Death (SCD) in Young Adults
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Sudden Cardiac Death (SCD) in Young Adults

A special issue of Journal of Clinical Medicine (ISSN 2077-0383). This special issue belongs to the section "Cardiology".

Deadline for manuscript submissions: closed (31 January 2024) | Viewed by 4614

Special Issue Editors

Dr. Paolo Di Donna

E-Mail Website
Guest Editor
IRCCS Policlinico San Martino, University Hospital of Genova, Genoa, Italy
Interests: cardiac genetic diseases; sudden cardiac death; inherited arrhythmia syndromes; electrophysiology; ablation and cardiac devices
Prof. Fiorenzo Gaita

E-Mail Website
Guest Editor
Department of Medical Sciences, University of Turin, Turin, Italy
Interests: cardiac genetic diseases; sudden cardiac death; inherited arrhythmia syndromes; electrophysiology; ablation and cardiac devices
Dr. Giuseppe Mascia

E-Mail Website
Guest Editor
IRCCS Policlinico San Martino, University Hospital of Genova, Genoa, Italy
Interests: cardiac genetic diseases; sudden cardiac death; inherited arrhythmia syndromes; electrophysiology; ablation and cardiac devices

Special Issue Information

Dear Colleagues,

Sudden cardiac death (SCD) in young adults is a rare event, but traumatic and today widely publicized. We invite you for a Special Issue entitled “Sudden cardiac death in young adults”. The aim of the Special Issue is to present current knowledge relevant to the topic, with special focus on areas of active research and controversy. We will consider research that may improve the prevention, identification, risk stratification, and treatment of subjects at greatest risk. We will also include genetic evaluation, post-mortem evaluation and identification of modifiable barriers to provide better outcomes after resuscitation of SCD in young adults. In this Special Issue, original research articles and reviews are welcome. We look forward to receiving your contributions.

Dr. Paolo Di Donna
Prof. Fiorenzo Gaita
Dr. Giuseppe Mascia
Guest Editors

Manuscript Submission Information

Manuscripts should be submitted online at www.mdpi.com by registering and logging in to this website. Once you are registered, click here to go to the submission form. Manuscripts can be submitted until the deadline. All submissions that pass pre-check are peer-reviewed. Accepted papers will be published continuously in the journal (as soon as accepted) and will be listed together on the special issue website. Research articles, review articles as well as short communications are invited. For planned papers, a title and short abstract (about 100 words) can be sent to the Editorial Office for announcement on this website.

Submitted manuscripts should not have been published previously, nor be under consideration for publication elsewhere (except conference proceedings papers). All manuscripts are thoroughly refereed through a single-blind peer-review process. A guide for authors and other relevant information for submission of manuscripts is available on the Instructions for Authors page. Journal of Clinical Medicine is an international peer-reviewed open access semimonthly journal published by MDPI.

Please visit the Instructions for Authors page before submitting a manuscript. The Article Processing Charge (APC) for publication in this open access journal is 2600 CHF (Swiss Francs). Submitted papers should be well formatted and use good English. Authors may use MDPI's English editing service prior to publication or during author revisions.

Keywords

  • channelopathies
  • cardiomyopathies
  • myocarditis
  • left ventricular scar
  • coronary artery abnormalities
  • congenital heart disease
  • genetics
  • ventricular arrhythmias
  • sudden cardiac death

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Published Papers (2 papers)

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14 pages, 1557 KiB  
Article
Electrocardiogram Changes in the Postictal Phase of Epileptic Seizure: Results from a Prospective Study
by Lorenzo Gigli, Simone Sala, Alberto Preda, Kenji Okubo, Giovanni Peretto, Antonio Frontera, Marisa Varrenti, Matteo Baroni, Marco Carbonaro, Sara Vargiu, Chiara Di Resta, Pasquale Striano, Patrizio Mazzone and Paolo Della Bella
J. Clin. Med. 2023, 12(12), 4098; https://doi.org/10.3390/jcm12124098 - 17 Jun 2023
Viewed by 2563
Abstract
Background: The brain and heart are strictly linked and the electrical physiologies of these organs share common pathways and genes. Epilepsy patients have a higher prevalence of electrocardiogram (ECG) abnormalities compared to healthy people. Furthermore, the relationship between epilepsy, genetic arrhythmic diseases and [...] Read more.
Background: The brain and heart are strictly linked and the electrical physiologies of these organs share common pathways and genes. Epilepsy patients have a higher prevalence of electrocardiogram (ECG) abnormalities compared to healthy people. Furthermore, the relationship between epilepsy, genetic arrhythmic diseases and sudden death is well known. The association between epilepsy and myocardial channelopathies, although already proposed, has not yet been fully demonstrated. The aim of this prospective observational study is to assess the role of the ECG after a seizure. Materials and Methods: From September 2018 to August 2019, all patients admitted to the emergency department of San Raffaele Hospital with a seizure were enrolled in the study; for each patient, neurological, cardiological and ECG data were collected. The ECG was performed at the time of the admission (post-ictal ECG) and 48 h later (basal ECG) and analyzed by two blinded expert cardiologists looking for abnormalities known to indicate channelopathies or arrhythmic cardiomyopathies. In all patients with abnormal post-ictal ECG, next generation sequencing (NGS) analysis was performed. Results: One hundred and seventeen patients were enrolled (females: 45, median age: 48 ± 12 years). There were 52 abnormal post-ictal ECGs and 28 abnormal basal ECGs. All patients with an abnormal basal ECG also had an abnormal post-ictal ECG. In abnormal post-ictal ECG, a Brugada ECG pattern (BEP) was found in eight patients (of which two had BEP type I) and confirmed in two basal ECGs (of which zero had BEP type I). An abnormal QTc interval was identified in 20 patients (17%), an early repolarization pattern was found in 4 patients (3%) and right precordial abnormalities were found in 5 patients (4%). Any kind modification of post-ictal ECG was significantly more pronounced in comparison with an ECG recorded far from the seizure (p = 0.003). A 10:1 higher prevalence of a BEP of any type (particularly in post-ictal ECG, p = 0.04) was found in our population compared to general population. In three patients with post-ictal ECG alterations diagnostic for myocardial channelopathy (BrS and ERP), not confirmed at basal ECG, a pathogenic gene variant was identified (KCNJ8, PKP2 and TRMP4). Conclusion: The 12-lead ECG after an epileptic seizure may show disease-related alterations otherwise concealed in a population at a higher incidence of sudden death and channelopathies. Post-ictal BEP incidence was higher in cases of nocturnal seizure. Full article
(This article belongs to the Special Issue Sudden Cardiac Death (SCD) in Young Adults)
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8 pages, 5140 KiB  
Case Report
A Soccer Shot with Lengthy Consequences—Case Report & Current Literature Review of Commotio Cordis
by Philipp Spitaler, Markus Stühlinger, Agne Adukauskaite, Axel Bauer and Wolfgang Dichtl
J. Clin. Med. 2023, 12(6), 2323; https://doi.org/10.3390/jcm12062323 - 16 Mar 2023
Cited by 1 | Viewed by 1588
Abstract
(1) Background: Commotio cordis, caused by objects being directly delivered to the chest, may cause cardiac arrest in young athletes, even without identifiable structural damage to the sternum, ribs or heart itself. Its prevention and management often remain suboptimal, resulting in dismal outcomes. [...] Read more.
(1) Background: Commotio cordis, caused by objects being directly delivered to the chest, may cause cardiac arrest in young athletes, even without identifiable structural damage to the sternum, ribs or heart itself. Its prevention and management often remain suboptimal, resulting in dismal outcomes. (2) Case summary: A 32-year semi-professional goalkeeper suffered from a non-penetrating blunt thoracic trauma after being struck by a high-velocity shot during a regional league soccer game. He immediately lost consciousness, collapsed, and was successfully resuscitated through early defibrillation of ventricular fibrillation. After an uneventful follow-up for approximately 6 years, recurrent episodes of ventricular tachycardia occurred, which could ultimately only be prevented by epicardial ablation. (3) Conclusion: Very late recurrences of ventricular tachyarrhythmias may occur after ventricular fibrillation due to blunt chest trauma, even in the primary absence of evident structural myocardial damage. Full article
(This article belongs to the Special Issue Sudden Cardiac Death (SCD) in Young Adults)
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