Mechanisms of Phosphate Transport
A special issue of International Journal of Molecular Sciences (ISSN 1422-0067). This special issue belongs to the section "Molecular Pathology, Diagnostics, and Therapeutics".
Deadline for manuscript submissions: closed (15 May 2021) | Viewed by 25127
Special Issue Editor
Special Issue Information
Dear Colleagues,
The International Journal of Molecular Science (IJMS, ISSN 1422-0067) has decided to publish an issue on the field of inorganic phosphate transport. This is an excellent, well-deserved opportunity because inorganic phosphate is involved not only in many physiological processes (buffer, macromolecule component, signal transduction, energy store, etc.) but also in the pathophysiology of processes caused by either the deprivation (hypophosphatemia), excess (hyperphosphatemia), or dysregulation (precipitation with calcium, deposition) of phosphate.
Over the last thirty years, in-depth knowledge on the identification of new Pi transporters and the corresponding molecular mechanisms of sodium–phosphate cotransport has been compiled. The study of interactions with associated proteins, of the hormonal and non-hormonal regulation of Pi transport, of transport-independent (paracellular) intestinal Pi absorption, and of toxicity during hyperphosphemia are just a few pending areas of research waiting to be studied in greater detail. Similarly, there is strong evidence that new Pi transporters exist, some of them functioning independently of sodium.
The Special Issue, “Mechanisms of phosphate transport”, of the International Journal of Molecular Science is intended to include original research papers and reviews about various aspects of the molecular and cellular biology of Pi transport. Studies on the molecular mechanisms of regulation and phosphate sensing and signaling are also welcomed, and pathophysiological studies that include the molecular mechanisms of diseases related to Pi dysregulation will also be considered.
Prof. Dr. Victor Sorribas
Guest Editor
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Keywords
- Pi
- Transporter
- NaPi
- PiT1
- PiT2
- Slc34
- Slc20
- Phosphatonins
- Pi homeostasis
- PTH
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