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Dietary Approach to Several Types of Primary and Secondary Dyslipidemias

A special issue of Nutrients (ISSN 2072-6643). This special issue belongs to the section "Lipids".

Deadline for manuscript submissions: closed (31 January 2024) | Viewed by 453

Special Issue Editor


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Guest Editor
1. Dietetics and Clinical Nutrition Unit, IRCCS Policlinic Hospital San Martino, 16132 Genoa, Italy
2. Department of Internal Medicine, University of Genoa, 16132 Genoa, Italy
Interests: clinical nutrition; clinical lipidology; genetics of dyslipidemias
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Special Issue Information

Dear Colleagues,

Dyslipidemias, defined as the alteration of at least one lipoprotein, are diseases correlated with cardiovascular risk and with the risk of acute pancreatitis in the case of hyperchylomicronemia. Lifestyle intervention is always recommended for the treatment of dyslipidemias. Some of them require targeted dietary therapy. In this Special Issue, we can focus on the optimal dietary interventions for the treatment of the main primary and secondary dyslipidemias such as mixed dyslipidemia in metabolic syndrome, renal diseases, hepatic disorders, hypercholesterolemia in heterozygous and homozygous familial hypercholesterolemia, sitosterolemia, hepatic steatosis and malabsorption in hypobetalipoproteinemia, severe hypertriglyceridemia with recurrent pancreatitis and the optimal diet for cardiovascular prevention. 

Dr. Livia Pisciotta
Guest Editor

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Keywords

  • diet and metabolic syndrome
  • diet and dyslipidemia and renal diseases
  • diet and dyslipidemia and hepatic disorders
  • diet and cardiovascular prevention
  • diet and hyperchilomicronemic syndrome
  • diet and familial monogenic hypercholesterolemia
  • diet and dysbetalipoproteinemia
  • diet and hypobetalipoproteinemia
  • diet in pediatric dyslipidemias
  • ketogenic diet and dyslipidemias
  • FMD and dyslipidemias
  • diet and dislipidemia in ederly

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Published Papers (1 paper)

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Review

29 pages, 1460 KiB  
Review
Characteristics, Physiopathology and Management of Dyslipidemias in Pregnancy: A Narrative Review
by Elena Formisano, Elisa Proietti, Giuseppina Perrone, Valentina Demarco, Paola Galoppi, Claudia Stefanutti and Livia Pisciotta
Nutrients 2024, 16(17), 2927; https://doi.org/10.3390/nu16172927 (registering DOI) - 1 Sep 2024
Abstract
Dyslipidemia is a significant risk factor for atherosclerotic cardiovascular disease (ASCVD). During pregnancy, physiological changes elevate cholesterol and triglyceride levels to support fetal development, which can exacerbate pre-existing conditions and lead to complications such as pre-eclampsia, gestational diabetes, and increased ASCVD risk for [...] Read more.
Dyslipidemia is a significant risk factor for atherosclerotic cardiovascular disease (ASCVD). During pregnancy, physiological changes elevate cholesterol and triglyceride levels to support fetal development, which can exacerbate pre-existing conditions and lead to complications such as pre-eclampsia, gestational diabetes, and increased ASCVD risk for both mother and child. Effective management strategies are necessary, especially for pregnant women with inherited forms of dyslipidemia (i.e., familial hypertriglyceridemia, hyperchylomicronemia), where personalized dietary adjustments are crucial for successful pregnancy outcomes. Pharmacological interventions and lipoprotein apheresis may be necessary for severe cases, though their use is often limited by factors such as cost, availability, and potential fetal risks. Despite the promise of advanced therapies, their widespread application remains constrained by limited studies and high costs. Thus, a personalized, multidisciplinary approach is essential for optimizing outcomes. This review provides a comprehensive overview of current strategies and evidence-based practices for managing dyslipidemia during pregnancy, emphasizing the balance of maternal and fetal health. Additionally, it discusses the physiological changes in lipid metabolism during pregnancy and their implications, particularly for women with inherited forms of dyslipidemia. Full article
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